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PreprintAll rights reservedbioRxiv

IL-23 licenses the transition from preclinical autoimmunity to inflammatory arthritis through IL-17A-dependent mechanisms

5 more authors
  1. 1Division of Rheumatology, Department of Medicine and Department of Pathology and Immunology, Geneva Center of Inflammation Research, Faculty of Medicine, Univer

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BibTeX
@misc{jarlborg2026il23,
  title = {{IL-23 licenses the transition from preclinical autoimmunity to inflammatory arthritis through IL-17A-dependent mechanisms}},
  author = {M. Jarlborg and A. Circiumaru and C. Lamacchia and L. Vaillant and E. Rakotoarijaonina and K. Venken and D. Elewaut and E. Rodriguez and A. Caruso and A. Huard and L. Santuari and N. Chammartin and D. S. Courvoisier and M. Andreatta and S. J. Carmona and A. Finckh and C. Gabay},
  howpublished = {bioRxiv},
  year = {2026},
  month = oct,
  doi = {10.64898/2026.10.30.762860},
  url = {https://www.biorxiv.org/content/10.64898/2026.10.30.762860v1},
}
RIS
TY  - UNPB
TI  - IL-23 licenses the transition from preclinical autoimmunity to inflammatory arthritis through IL-17A-dependent mechanisms
AU  - M. Jarlborg
AU  - A. Circiumaru
AU  - C. Lamacchia
AU  - L. Vaillant
AU  - E. Rakotoarijaonina
AU  - K. Venken
AU  - D. Elewaut
AU  - E. Rodriguez
AU  - A. Caruso
AU  - A. Huard
AU  - L. Santuari
AU  - N. Chammartin
AU  - D. S. Courvoisier
AU  - M. Andreatta
AU  - S. J. Carmona
AU  - A. Finckh
AU  - C. Gabay
PY  - 2026
DA  - 2026/10/30
PB  - bioRxiv
DO  - 10.64898/2026.10.30.762860
UR  - https://www.biorxiv.org/content/10.64898/2026.10.30.762860v1
ER  -