PreprintAll rights reservedbioRxiv
Mitochondrial transplantation redirects rather than suppresses the inflammatory response in primary human monocytes
- 1Massachusetts General Hospital
Related
- Complex I Inhibition Redirects Autophagy from Bulk Turnover to Mitochondrial Clearance
- The ability of SAMHD1-deficient monocytes to trigger the Type I IFN response depends on cGAS and mitochondrial DNA
- A TLR2-associated entry pathway promotes productive dengue virus serotype 2 infection and inflammatory responses in classical monocytes
- Tirzepatide preserves hematopoietic stem and progenitor cycling while remodeling inflammatory monocytes in obese mice
- AMPK reinforces mitochondrial metabolism and suppresses pathological remodeling in Complex V-deficient cardiomyocytes
BibTeX
@misc{nazemidashtarjandi2026mitochondrial,
title = {{Mitochondrial transplantation redirects rather than suppresses the inflammatory response in primary human monocytes}},
author = {S. Nazemidashtarjandi and S. Pantham and R. Dori and D. Bin Back and G. Hamanaka and K. Hawakaya and M. Yarmush},
howpublished = {bioRxiv},
year = {2026},
month = nov,
doi = {10.64898/2026.11.06.762873},
url = {https://www.biorxiv.org/content/10.64898/2026.11.06.762873v1},
}
RIS
TY - UNPB
TI - Mitochondrial transplantation redirects rather than suppresses the inflammatory response in primary human monocytes
AU - S. Nazemidashtarjandi
AU - S. Pantham
AU - R. Dori
AU - D. Bin Back
AU - G. Hamanaka
AU - K. Hawakaya
AU - M. Yarmush
PY - 2026
DA - 2026/11/06
PB - bioRxiv
DO - 10.64898/2026.11.06.762873
UR - https://www.biorxiv.org/content/10.64898/2026.11.06.762873v1
ER -