PreprintAll rights reservedbioRxiv
Rethinking Syndecan-1 as a Marker of Trauma-Induced Endothelial Injury
- 1University of California, San Francisco
Related
- Loss of Syndecan-3 Drives a Pro-inflammatory, EndMT-prone Phenotype in Aortic Valve Endothelial Cells
- Syndecan-3 couples tumor mechanics to immunosuppressive macrophage identity
- Human organ chip models recapitulate FRC-driven high endothelial venule differentiation, immune recruitment, and cancer-induced disruption
- Reproducible detection of antigen-specific T cells and Tregs via standardized and automated activation-induced marker assay workflows
- Bottom‐Up Engineering of a Human Neuromuscular System for Modeling Activity‐Induced Remodeling, Metabolic Stress, and Endothelial‐Modulated Excitability
BibTeX
@misc{pati2026rethinking,
title = {{Rethinking Syndecan-1 as a Marker of Trauma-Induced Endothelial Injury}},
author = {S. Pati and H. Geng and A. Trivedi and C. Keane and M. Barry and P. Pathipati and K. A. Bertram and E. E. Moore and M. Schreiber and J. Cuschieri and J. B. Holcomb and E. Zacco and A. B. Nair},
howpublished = {bioRxiv},
year = {2026},
month = oct,
doi = {10.64898/2026.10.12.758409},
url = {https://www.biorxiv.org/content/10.64898/2026.10.12.758409v1},
}
RIS
TY - UNPB
TI - Rethinking Syndecan-1 as a Marker of Trauma-Induced Endothelial Injury
AU - S. Pati
AU - H. Geng
AU - A. Trivedi
AU - C. Keane
AU - M. Barry
AU - P. Pathipati
AU - K. A. Bertram
AU - E. E. Moore
AU - M. Schreiber
AU - J. Cuschieri
AU - J. B. Holcomb
AU - E. Zacco
AU - A. B. Nair
PY - 2026
DA - 2026/10/12
PB - bioRxiv
DO - 10.64898/2026.10.12.758409
UR - https://www.biorxiv.org/content/10.64898/2026.10.12.758409v1
ER -