Peer-reviewedCC BY-NC-NDInternal Medicine
Response to "The Afatinib Response to EGFR E709G-mutated Lung Cancer"
Related
- Response to Letter to the Editor on "The Afatinib Response to EGFR E709G-mutated Lung Cancer"
- Venetoclax plus hypomethylating agent improves response rates but not survival in TP53-mutated myelodysplastic syndromes.
- Integrated stress response activation in combination PG3 and cisplatin-treated TP53-mutated head and neck squamous carcinoma cells induces potent apoptotic response
- Genetic dissection of DNA damage response pathways reveals divergent modulation of PD-1 blockade response in colorectal cancer
- Pan-Cancer Gene Expression Predicts Per-Cancer Radiation Therapy Response
BibTeX
@article{isaac2026response,
title = {{Response to "The Afatinib Response to EGFR E709G-mutated Lung Cancer"}},
author = {Sangeetha Isaac and Misbah Qadir},
journal = {Internal Medicine},
year = {2026},
month = jul,
doi = {10.2169/internalmedicine.5658-25},
url = {https://www.jstage.jst.go.jp/article/internalmedicine/65/14/65_5658-25/_article},
note = {CC BY-NC-ND},
}
RIS
TY - JOUR
TI - Response to "The Afatinib Response to EGFR E709G-mutated Lung Cancer"
AU - Sangeetha Isaac
AU - Misbah Qadir
PY - 2026
DA - 2026/07/15
JO - Internal Medicine
DO - 10.2169/internalmedicine.5658-25
UR - https://www.jstage.jst.go.jp/article/internalmedicine/65/14/65_5658-25/_article
SN - 1349-7235
SN - 0918-2918
ER -