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Peer-reviewedCC BY-NC-ND 4.0Virologica Sinica

ACSL3 at the UPR crossroads: Tactician of anti-influenza defense, target of influenza offense

  • Qiong Mo1
  • Yun-Jia Ning2,3,4
4 affiliations
  1. 1Department of Infectious Diseases, The Affiliated People's Hospital of Ningbo University, Ningbo 315020, China
  2. 2State Key Laboratory of Virology and Biosafety, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan 430071, China
  3. 3University of Chinese Academy of Sciences, Beijing 101408, China
  4. 4Hubei Jiangxia Laboratory, Wuhan 430200, China
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Abstract

Highlights: •A new dimension of the host-IAV arms race is revealed, centered on ACSL3 at the UPR crossroads.

•Key questions remain regarding ACSL3-UPR sensor interfaces and ACSL3 induction/degradation mechanisms.

•Addressing the questions will advance ACSL3-IAV interplay insights and may inform host-targeted therapy.

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Cite

BibTeXRIS
BibTeX
@article{mo2026acsl3,
  title = {{ACSL3 at the UPR crossroads: Tactician of anti-influenza defense, target of influenza offense}},
  author = {Qiong Mo and Yun-Jia Ning},
  journal = {Virologica Sinica},
  year = {2026},
  month = sep,
  doi = {10.1016/j.virs.2026.09.002},
  url = {https://doi.org/10.1016/j.virs.2026.09.002},
  note = {CC BY-NC-ND 4.0},
}
RIS
TY  - JOUR
TI  - ACSL3 at the UPR crossroads: Tactician of anti-influenza defense, target of influenza offense
AU  - Qiong Mo
AU  - Yun-Jia Ning
PY  - 2026
DA  - 2026/09/04
JO  - Virologica Sinica
DO  - 10.1016/j.virs.2026.09.002
UR  - https://doi.org/10.1016/j.virs.2026.09.002
AB  - Highlights: •A new dimension of the host-IAV arms race is revealed, centered on ACSL3 at the UPR crossroads.  •Key questions remain regarding ACSL3-UPR sensor interfaces and ACSL3 induction/degradation mechanisms.  •Addressing the questions will advance ACSL3-IAV interplay insights and may inform host-targeted therapy.
SN  - 1674-0769
SN  - 1995-820X
ER  -